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A drug used to treat chronic myeloid leukemia appears to be more effective at stopping a type of medulloblastoma in mouse models than existing treatments for the deadly pediatric brain tumor, reports a multi-institutional team led by researchers at Skaggs School of Pharmacy and Pharmaceutical Sciences at University of California San Diego.
The outer layer of the skin completely replaces itself every two to four weeks, but when this process is blocked, cancer can grow.
Diane and Phil Hannah started their lifelong journey together as neighbors who would talk to each other from their bedroom windows. They were high school sweethearts before they married in 1952.
Under the leadership of Vishal A. Patel, MD, FAAD, FACMS, director of the Cutaneous Oncology Program at the George Washington University Cancer Center, the GW Cancer Center was selected as the first global site for a clinical trial for patients with high-risk cutaneous squamous cell carcinoma.
Skin cancer is the most common cancer in the U.S., affecting one in five Americans in their lifetime. Limiting exposure to ultraviolet radiation is the number one way individuals can reduce their risk of skin cancer, though new data suggests that UV exposure is on the rise, particularly among Caucasian girls and young women.
It is a well known fact that the sun’s ultraviolet rays can damage the skin. Now, a new study sheds light on what happens after DNA damage occurs, and how the body initiates repair.
Sun safety practices for attendees at skin cancer screening events differ from the general public, according to findings published by researchers from the George Washington University Cancer Center.
Whenever you're outdoors this summer, be sure to take steps to prevent solar erythema, the acute cutaneous reaction to excessive exposure to ultraviolet (UV) radiation.
When it comes to the diagnosis of pigmented skin lesions, artificial intelligence is superior to humans. In a study conducted under the supervision of the MedUni Vienna human experts "competed" against computer algorithms.
Differential H3K27AC marks were identified at enhancer regions of genes including c-MYC, MED1, OCT-4, NANOG, and SOX2 that can act as super-enhancers (SE), cluster of genes in non-cancerous, cancerous and metastatic PDAC cell lines.